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It significantly ameliorated AA-induced tubular necrosis and interstitial fibrosis by up-regulating the renal expression of Nrf2 and Smad7 (Song et al., 2019)

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Figure 2 3.2 Metabolic reprogramming in M1 macrophages Glycolysis is a crucial metabolic event for M1 macrophages, and its inhibition affects typical functions of their inflammatory phenotype, including phagocytosis, ROS production, and pro-inflammatory cytokine secretion