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Integrins such as α5β1, αvβ3, and α1β1 bind ECM ligands (e.g., collagen type II, fibronectin) and cluster at focal adhesions, where focal adhesion kinase (FAK) initiates downstream effectors like the Rho GTPases (RhoA, Rac1) and MAPKs (e.g., ERK1/2, p38)

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Activating or shutting off multiple signal pathways by affecting transcription factors including NF-κB, MYC, FOS, CREB, WT1, and mTOR may also be the reason for its anti-tumor effects