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GSH is known to cleave disulfide and diselenide bonds by binding to one cleaved thiol/selenol, forming another disulfide or sulfide-selenide linkage, liberating the other half of disulfide/diselenide as a thiol/selenol, thus being an efficient reducing agent

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Current treatments for PD are primarily symptomatic, targeting dopamine synthesis or metabolism through LD, COMT inhibitors, and MAO-B inhibitors