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In the absence of thromboxane-prostanoid receptors, PGI2/iloprost treatment stimulates cell proliferation, upregulates synthetic proteins, downregulates contractile proteins, and promotes VSMC phenotypic switching [91]

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In contrast, loss of p53 prevents the accumulation of dipeptidyl-peptidase-4 (DPP4) in the nucleus, triggering membrane-associated DPP4-mediated lipid peroxidation