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It has been shown to modulate redox-sensitive transcription factors such as Nrf2 (nuclear factor erythroid 2-related factor 2) and NF-κB (nuclear factor kappa B) [37,38,39], thereby simultaneously enhancing endogenous antioxidant defenses and suppressing pro-inflammatory signaling [40,41,42]

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As DFX treatment reduced iron levels, decreased cell death, and promoted myelination, we hypothesized that DFX treatment would affect the enzymes involved in regulating iron, lipid, and oxylipin/eicosanoid metabolism